Biological mechanism of turbocancer
Scientists had already raised the alarm during the first vaccination campaigns in 2021. Among them, Jean-Marc Sabatier, a doctor of cell biology and microbiology and research director at the CNRS, documented a possible link between mRNA vaccines and the development of rapidly progressing tumors, the so-called turbocancers.
According to his explanation, this phenomenon results from excessive activation of the AT1R receptor in the renin-angiotensin system. This system plays a key role in cell differentiation and proliferation. The spike protein produced after vaccination binds to the ACE2 receptor, thus halting the degradation of angiotensin II. In excess, this causes overstimulation of the AT1R receptor, which has pro-tumor, pro-angiogenic, and pro-inflammatory properties.
The AT1R receptor also promotes oxidative stress, releasing free radicals that are harmful to cells. In turn, antagonists of this receptor have been shown to have antitumor effects in various cancers, including breast, prostate, and lung cancer. Jean-Marc Sabatier described in March 2020, well before the first cases were reported, a carcinogenic effect resulting from the alteration of this system by the spike protein of the SARS-CoV-2 virus.
